CONTROVERSIAL TOPIC

What is the Bilderberg Group? - The Illuminati - (Full Documentary)
Published on 14 Dec 2014
What is the Bilderberg Group and who are they? Watch this great documentary and find out who they are, what they do and how does it affect everyone else. 
https://www.youtube.com/watch?v=xkmhmOKDlL4

http://www.disclose.tv/news/dying_cia_agent_claims_he_assassinated_marilyn_monroe_and_36_more_targets/116887
DYING CIA AGENT CLAIMS HE ASSASSINATED MARILYN MONROE, AND 36 MORE TARGETS 34.3K Normand Hodges swears he remembers vividly, each of the assassinations he committed for the CIA.
 He claims that all 37 of the murders he committed on the American soil, were ordered by his commanding officer, Major James “Jimmy” Hayworth. (Photo via World News Daily) Dying CIA officer Normand Hodges claims he assassinated 37 people, including Marilyn Monroe. 
The 78-year-old said he worked for the CIA for 41 years as an operative. He also admitted to acting as a hitman for the governmental agency. Normand Hodges is slowly dying. 
The retired CIA officer confessed to committing the assassinations during his stay at Sentara Norfolk General Hospital, in Norfolk, Virginia, according to a World News Daily report. Hodges was trained in martial arts and as a sniper. 
However, the former CIA agent said he is well versed in using explosives and poisons. 
MARILYN MONROE'S DEATH CONSPIRACY 
According to Hodges, Marilyn Monroe was the only woman he ever assassinated. Hodges said Marilyn “had to die.” Monroe had to be eliminated because the famed actress had become a “threat for the security of the country.” RELATED ARTICLES Top secret CIA poison dart gun 'gives targets deadly HEART ATTACKS' Rumor: Kim Jong Un Assassinated Early Friday Morning Your News Wire reported a comment made by Normand Hodges regarding Marilyn Monroe’s death. 
“We had evidence that Marilyn Monroe had not only slept with Kennedy, but also with Fidel Castro. My commanding officer, Jimmy Hayworth, told me that she had to die, and that it had to look like a suicide or an overdose. 
I had never killed a woman before, but I obeyed orders … I did it for America! She could have transmitted strategic information to the communists, and we couldn’t allow that! She had to die! I just did what I had to do!” MARILYN MONROE DIED AT HOME Marilyn Monroe died at home in her bedroom on August 5, 1962 between midnight and 1:00 AM. Hodges said that, while Marilyn was asleep, he snuck into her bedroom and injected the Hollywood actress with a huge dose of the barbiturate, Nembutal, combined with a powerful sedative known as chloral hydrate. Hodges said he and four other CIA operatives carried out political assassinations across the country. 
According to Normand, the majority of the victims were union leaders, political activists, and journalists. 
However, he confessed that some scientists and artists, whose thoughts and actions threatened the interests of the United States, were also on the hit list. WAS THE CIA INVOLVED IN MONROE'S DEATH? 
Four of the CIA operatives that worked in conjunction with Hodges are now dead or missing. In 2011, Normand’s commanding officer, Major James Hayworth, died of a heart attack. 
Two other CIA assassins are also dead, and the last one, Captain Keith McInnis, is presumed dead after being reported missing in action in 1968. The FBI is not taking Normand Hodges’ confessions lightly. They have placed the former CIA agent under custody and are conducting an investigation. 
However, as one might assume, very little documentation is available pertaining to such clandestine operations, including the death of Marilyn Monroe ( via inquisitr.com ). 
READ MORE: http://www.disclose.tv/news/dying_cia_agent_claims_he_assassinated_marilyn_monroe_and_36_more_targets/116887

http://www.nytimes.com/2016/02/23/science/scientists-ponder-the-prospect-of-contagious-cancer.html?smid=fb-share
Scientists Ponder the Prospect of Contagious Cancer
 George Johnson
For all its peculiar horror, cancer comes with a saving grace. If nothing else can stop a tumor’s mad evolution, the cancer ultimately dies with its host. Everything the malignant cells have learned about outwitting the patient’s defenses — and those of the oncologists — is erased. The next case of cancer, in another victim, must start anew.

Imagine if instead, cancer cells had the ability to press on to another body. A cancer like that would have the power to metastasize not just from organ to organ, but from person to person, evolving deadly new skills along the way.

While there is no sign of an imminent threat, several recent papers suggest that the eventual emergence of a contagious human cancer is in the realm of medical possibility. This would not be a disease, like cervical cancer, that is set off by the spread of viruses, but rather one in which cancer cells actually travel from one person to another and thrive in their new location.

So far this is known to have happened only under the most unusual circumstances. A 19-year-old laboratory worker who pricked herself with a syringe of colon cancer cells developed a tumor in her hand. A surgeon acquired a cancer from his patient after accidentally cutting himself during an operation. There are also cases of malignant cells being transferred from one person to another through an organ transplant or from a woman to her fetus.

On each of these occasions, the malignancy went no further. The only known cancers that continue to move from body to body, evading the immune system, have been found in other animals. In laboratory experiments, for instance, cancer cells have been transferred by mosquitoes from one hamster to another. And so far, three kinds of contagious cancers have been discovered in the wild — in dogs, Tasmanian devils and, most recently, in soft shell clams.

The oldest known example is a cancer that spreads between dogs during sexual intercourse — not as a side effect of a viral or bacterial infection, but rather through direct conveyance of cancer cells. The state of the research is described in a review, “The Cancer Which Survived,” published last year by Andrea Strakova and Elizabeth P. Murchison of the University of Cambridge.

The condition, canine transmissible venereal tumor disease, is believed to have sprung into existence 11,000 years ago — as a single cell in a single dog — and has been circulating ever since. (Why did this happen in dogs and not, say, cats? Perhaps because of what the authors demurely call the dogs’ “long-lasting coital tie” — the half an hour or so that a male and female are locked in intercourse, tearing genital tissues and providing the cancer cells with a leisurely crossing.)

Normally a cancer evolves in a single body over the course of years or decades, accumulating the mutations that drive it to power. But to have survived for millenniums, researchers have proposed, canine cancer cells may have developed mechanisms — like those in healthy cells — to repair and stabilize their own malignant genomes.

Early on, cancer cells typically flourish by disabling DNA repair and ramping up the mutational frenzy. Somewhere along the way, the age-old canine cells may have reinvented the device to extend their own longevity. There is also speculation that this cancer may have learned to somehow modify canine sexual behavior in ways that promote the disease’s spread and survival.

The second kind of contagious cancer was discovered in the mid-1990s in Tasmanian devils, which spread malignant cells as they try to tear off one another’s faces. Though it may be hard to sympathize, devil facial tumor disease threatens the creatures with extinction.

With so few examples, transmissible cancer has been easy to dismiss as an aberration. But in December, scientists at the Universities of Tasmania and Cambridge reported in Proceedings of the National Academy of Sciences that Tasmanian devils are passing around another kind of cancer — genetically distinct from the first. It’s weird enough that one such cancer would arise in the species. What are the chances that there would be two?

One theory is that the animals are unusually vulnerable. Driven so close to extinction — by climate change, perhaps, or human predators — the species is lacking in genetic diversity. The cells of another devil injected through a vicious wound may seem so familiar that they are ignored by the recipient’s immune system. If some of the cells carry the mutations for the facial cancer, they might be free to flourish and develop into a new tumor.

But the scientists also proposed a more disturbing explanation: that the emergence of contagious cancer may not be so rare after all. “The possibility,” they wrote, “warrants further investigation of the risk that such diseases could arise in humans.”

Cancer has probably existed ever since our first multicellular ancestors appeared on Earth hundreds of millions of years ago. The life spans of even the longest-lived animals may be just too brief for cancers to easily evolve the ability to leap to another body. Otherwise, contagious cancer would be everywhere.

For now, at least, it remains a curiosity. Consider the case of a 41-year-old man in Medellin, Colombia, who was examined by doctors in 2013 because of fatigue, fever and weight loss. His lymph nodes were clogged with cancer cells that had also spread to his lungs and liver.

Yet the cells looked far too small and simple to be human. “This case posed a diagnostic conundrum,” the doctors wrote in November in The New England Journal of Medicine.

The solution to the puzzle came when the man was also found to be harboring a tapeworm called Hymenolepis nana. Further analysis concluded that the cancer cells had originated in the parasite and then metastasized through the man’s body.

There is no reason to think that tapeworm cancer is about to become a threat to public health. The patient’s immune system had been compromised by H.I.V., and he died several months later.

But nature is infinite in its surprises.

Correction: February 22, 2016 
An earlier version of this article misstated one of the animals in which contagious cancer has been discovered in the wild. It was in soft shell clams, not crabs.
A version of this article appears in print on February 23, 2016, on page D3 of the New York edition with the headline: Scientists Ponder Contagious Cancer.